Deep, qualified following-era sequencing has uncovered that subclonal mutations (i.e., Individuals present in just a portion of tumor cells) is usually detected for all driver genes and so are linked to fast disease development and bad outcome.eleven–13 This is particularly applicable for TP53
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gene in individuals relapsing after treatment with the BCL2 antagonist venetoclax. 66 Resistance to those brokers has been connected to these mutations in all around 70% of situations, although they tend to be subclonal and their unique function causing resistance has to be demonstrated.
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